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The Skin Flint Podcast

The Skin Flint Podcast

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Whether you simply have a pet with skin issues, or are a vet / vet nurse looking to bolster your CPD record with free, easy to listen to, on the go discussion on and around pet skin disease - this is the podcast for you! Join European leading dermatologist Dr Sue Paterson, Dermatology Veterinary Nurse John Redbond and Elearning.Vet content provider Paul Heasman as they pick their way through the scabby surface of pet skin disease. Expect interviews with some of the smartest minds in animal dermatology to get beneath the surface of the latest thinking on all things fur and skin, keeping their gloved fingers on the pulse of current topics itching to be discussed. This podcast is brought to you by Nextmune UK (formerly Vetruus), specialist in veterinary dermatology and immunotherapy. Nextmune bring you products such as Otodine and CLX Wipes – market leading products in the management of skin and ear cases. In association with Elearning.Vet - providing the highest quality veterinary content free of charge.Copyright 2021 All rights reserved. Science
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    Épisodes
    • Episode 36 - Hives, Lesions and Lumps; Urticaria in Horses
      Jan 7 2026
      Episode Overview Join us as we venture into the stables to explore urticaria in horses - those mysterious swellings that appear seemingly out of nowhere and may disappear just as suddenly. Expert guest Dr. Valerie Fadok shares her extensive experience as both a veterinary dermatologist and immunologist to help us understand what causes these puzzling conditions, how to differentiate them from other lumps, and when to investigate further rather than automatically reaching for steroids. Featured Guest Dr. Valerie Fadok - A dual specialist bringing unique expertise as both a veterinary dermatologist and immunologist. With experience across three veterinary schools, private practice, and as a field specialist with Zoetis, Val brings a wealth of practical knowledge from working with veterinarians and horse owners around the world. Episode Breakdown Introduction to Urticaria in Horses Val discusses how horses are the most commonly affected species with urticaria among the animals veterinarians treat, and how this condition can drive both horses and their owners to distraction. The disease presents unique challenges, with sudden onset cases that sometimes resolve on their own, and chronic cases where horses experience repeated outbreaks over time. Clinical Presentation and Diagnosis What Urticaria Looks Like: Val emphasizes the importance of palpation—urticarial lesions tend to be soft compared to nodular diseases like eosinophilic granulomasIndividual lesions wax and wane, even if the horse has hives every dayLesions can take fascinating shapes: round, linear, or ring-like configurations (serpiginous patterns)Not all horses with urticaria are particularly itchy Papular Urticaria: Papular (miliary) lesions are commonly associated with insect bitesVal shares examples of horses moving from northern US states to Florida developing papular urticaria in their first year due to high insect pressure from mosquitoes and CulicoidesThese cases often resolve after the first yearSue confirms similar patterns in the UK with Culicoides Immunological vs Non-Immunological Reactions The Role of Mast Cells: Urticaria involves mast cells in the skinImmunological urticaria occurs when allergens bind to IgE on mast cells, triggering the reactionNon-immunological causes involve "twitchy" mast cells that react to physical triggers Physical Urticaria: Pressure urticaria and dermatographism—where a handprint appears on the horse's flank after touchingCold-induced urticariaHeat-induced urticariaExercise-induced urticariaSome horses have both immunological and physical components, making diagnosis particularly challenging History is Key: Observant owners can provide crucial information (e.g., "hives appeared after training session" or "outline of saddle appeared after removal")Owner observations are often the best way to differentiate between causes Acute vs Chronic Urticaria Acute Urticaria Management: Most acute urticaria in horses is drug-related (antibiotics, pain medications) or from blood transfusionsVal's approach: Don't do an intense workup immediatelyTreat with antihistamines (Val prefers hydroxyzine) for a few months to let mast cells settleIf it recurs after stopping medication, then investigate furtherSue agrees: not chronic unless present for 8+ weeks or recurring annually When to Investigate: Sue and Val agree: 8-12 weeks or recurrent episodes warrant deeper investigationBoth emphasize the value of owners who keep detailed calendars noting when hives appear50% of urticaria in people remains idiopathic—same often true for horsesCompetition horses present particular challenges due to medication restrictions Investigation and Testing Seasonal Cases: For seasonal urticaria, Val recommends intradermal or serum allergy testingHorses with urticaria respond well to allergen immunotherapy compared to other speciesMost horse owners are comfortable giving injections Non-Seasonal Cases: Consider dietary factors and whether feed changes throughout the yearHorse owners are surprisingly open to food trialsVal has only proven a handful of food-related urticaria cases (alfalfa and grains)Diet trials are difficult in horses, though owners are willing Environmental Allergens: House dust mites and storage mites are the most commonly identified allergens across all speciesMolds are important triggers, especially in humid environmentsVal notes regional differences: Florida has unusual pollens and insects, Texas is drier with mainly pollens, Pacific Northwest sees more mold allergiesSue observes autumn cases in UK when horses start wearing rugs, potentially related to house dust mites, temperature, dampness, or molds Allergen-Specific Immunotherapy Val's Approach: Uses traditional step-up procedure for injection immunotherapyConsults pollen charts (from Greer allergy company, pollen.com, Google searches)Selects major allergens relevant to the horse's region and historyDoesn't include everything that tests positive—focuses on major ...
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      38 min
    • Episode 35 - Ferrets, Fur and Fun; Taming The Beastmasters
      Nov 29 2025

      In this month's episode, Sue John and Paul invite exotics guru Madonna Livingstone onto the podcast to discuss general and skin issues in ferrets.

      Overview of ferrets as pets:

      - Ferrets have been domesticated for over 3,000 years, evolving from the European polecat.

      - They are highly social and prefer to be kept in groups of at least two.

      - Ferrets have high protein and fat dietary requirements, and need a lot of space and environmental enrichment.

      Common Skin Conditions in Ferrets:

      - Ectoparasites:

      - Fleas (usually dog and cat fleas) are the most common ectoparasite, causing pruritus and alopecia.

      - Ear mites are very common in ferrets, often asymptomatic.

      - Sarcoptic mange can affect the feet, causing severe pruritus and scabs.

      - Allergies:

      - Skin allergies are rare in ferrets, but environmental contact dermatitis can occur.

      - Food allergies are very uncommon.

      - Hormonal Imbalances:

      - Hyperadrenocorticism (adrenal gland disease) is less common than hyperestrogenism in female ferrets.

      - Hyperestrogenism can lead to alopecia, bone marrow suppression, and even death if untreated.

      Treatment Approaches:

      - Ectoparasites: Use of selamectin, fipronil, or moxidectin is recommended.

      - Allergies: Antihistamines, steroids, and omega-3/6 fatty acids can be used.

      - Hormonal Imbalances:

      - Hyperestrogenism is often managed with deslorelin implants or HCG injections.

      - Hyperadrenocorticism may require surgical adrenalectomy or deslorelin implants.

      Insights and Takeaways

      - Ferrets are unique and fascinating pets, with specific care and medical needs.

      - Skin conditions in ferrets can present similarly to dogs and cats, but there are some key differences in diagnosis and treatment.

      - Ectoparasites, especially fleas and ear mites, are very common and should be the first consideration when a ferret presents with skin disease.

      - Hormonal imbalances, particularly hyperestrogenism in females, are an important cause of skin problems in ferrets and require prompt recognition and management.

      - Veterinarians should be familiar with ferret-specific care and be prepared to handle ferret skin cases, as they may be uncommon but can be challenging.

      Conclusions and Decisions

      - Ferrets are becoming increasingly popular pets, and veterinarians should be equipped to provide appropriate care for their skin conditions.

      - Developing a good understanding of ferret biology, common skin diseases, and treatment approaches can help build confidence in managing these cases.

      - Consulting resources like the BSAVA Exotic Animal Formulary and seeking advice from experienced exotic animal veterinarians can be invaluable when caring for ferrets with skin problems.

      - Promoting proper husbandry and preventive care, such as regular ectoparasite control and monitoring for hormonal imbalances, can help minimize skin issues in ferrets.

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      49 min
    • Episode 34 - Short Noses, Big Problems: Lumps, Chins and Comorbidities in Brachycephalics
      Nov 3 2025
      Chapter 1 – “Lumps, Not Bugs: Cracking the ‘Sterile’ Case” 03:22 – John welcomes Dr Laura Buckley back for part two on brachycephalic skin disease and tees up two topics: Sterile Granuloma/Pyogranuloma Syndrome (SGPS) and Muzzle Folliculitis/Furunculosis, plus how to manage comorbidities. Laura explains it’s an uncommon, immune‑mediated nodular skin disease of dogs involving histiocytic cells (macrophages). No infectious agent is found and it responds to immunomodulatory therapy. 04:43 – Sue asks which brachy breeds are most affected and typical ages. Laura most often sees Boxers, British Bulldogs, some Mastiffs and (in her clinic) many Staffordshire Bull Terriers. Usual onset is middle‑aged, though younger dogs can be affected. 05:21 – Sue asks what it looks like. Laura: papules, nodules or plaques (mm to several cm), localised or generalised; often on trunk, but head/limbs too. Typically non‑painful and non‑pruritic; may be erythematous, haired or alopecic; sometimes eroded/ulcerated with crusting - the key is a nodular process. 07:20 – Sue asks for key differentials. Laura highlights superficial bacterial folliculitis as the big rule‑out in short‑coated brachys (tufted hairs). Cytology helps: infection shows neutrophils with intracellular cocci (staphylococci); a sterile process shows inflammatory cells without bacteria. 07:49 – Laura notes most SGPS nodules are intact, so fine‑needle aspirates (multiple nodules) are preferred over impression smears. Expect many neutrophils and macrophages; bacteria should be absent. 08:23 – Sue asks about deep fungal disease and other infections. Laura: you can’t reliably exclude on cytology alone—next step is biopsy. Remove a whole nodule if possible so histopathology can section through it and use special stains for atypical organisms (bacteria, deep fungi, parasites, protozoa). This thorough exclusion is critical before immunosuppression. 10:11 – Sue asks how to submit samples. Laura often splits: submit an entire nodule (or half) in formalin for histopathology and keep a second small sample (e.g., 4 mm punch from another lesion) chilled/frozen pending culture. Direct to bacteriology or mycology depending on histopath hints. 11:01 – John asks about treatment and prognosis. Laura finds most dogs do well: disease may wax and wane but responds to therapy; rare spontaneous resolution reported. Start with glucocorticoids (prednisolone). Typical immunosuppressive dose 2–4 mg/kg (sometimes 1–1.5 mg/kg suffices; she often starts at 2 mg/kg). If response is poor or steroid side effects are problematic, add cyclosporine at 5 mg/kg once daily; azathioprine has been used. For localised lesions, topical hydrocortisone aceponate spray can help. 13:24 – Sue asks for a prednisolone protocol. Laura: baseline haematology/biochemistry/urinalysis before starting. Recheck at 2–3 weeks for tolerance and early response; continue same dose another 2–3 weeks to resolution, then taper by ~20% every couple of weeks. Once down to ~0.5 mg/kg, move to alternate‑day dosing. Add cyclosporine if lesions recur on taper to avoid long‑term steroid adverse effects (PU/PD/PP, lethargy/weight gain; long‑term risk: calcinosis cutis). With dual therapy or cytotoxics, schedule regular bloods (after 1 month, then every 2–3 months). Chapter 2 – “Chins Up: Muzzle Mayhem, Managed” 17:05 – John pivots to Muzzle Folliculitis/Furunculosis: what is it and who gets it? Laura: a bacterial follicular disease confined to the muzzle skin, common in coarse/bristly‑coated brachys—British & French Bulldogs, Pugs, Shar‑Pei, Boxers. 18:04 – John asks what drives it. Laura: often linked to allergic skin disease; facial folds create many “mini‑intertrigo” sites. Pruritus → rubbing/trauma to bristly follicles. She suspects a sterile inflammatory start that quickly progresses to secondary bacterial folliculitis. 20:13 – Sue asks if this is the same as acne. Laura: no - acne is a keratinisation disorder (e.g., plugged follicles; classic in cat chins). Muzzle folliculitis/furunculosis is follicular inflammation progressing to follicle rupture (furunculosis) with foreign‑body reaction. Clinically it’s more diffuse over chin/muzzle with erythema, alopecia, papules/pustules, erosions/ulcers/crusts; severe cases show haemorrhagic bullae‑like lesions—“an interdigital cyst on the chin.” 23:24 – John asks about diagnosis and first‑line management. Laura: clinical pattern + cytology to confirm/grade infection. Prioritise topical antiseptics; address primary disease and contributing behaviours (chewing cages/toys, environment). Systemic antibiotics only if deep/severe infection and ideally based on culture. 25:02 – Sue asks preferred topicals. Laura: chlorhexidine‑containing products are mainstay; ethyl lactate also helpful. Choose gentle vehicles (mousses/wipes) for faces; shampoos are good for debris removal ...
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      37 min
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